• @ColeSloth@discuss.tchncs.de
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    188 months ago

    It turns out in 1961 the American heart Association took bribery money from procter and gamble, who owned and sold “healthier Crisco” cooking oils that weren’t high in saturated fat, like beef and other cooking oils were.

    The AHA then claimed and pushed that saturated fats caused heart disease.

    Problem is, something like 88% of every study done in the past 60 years has found little to no link between heart disease and saturated fats.

    So beef, according to most studies, isn’t bad for you. The AHA was just crooked and on the take, being paid off to sell Crisco.

    Now it is calorie dense and people tend to eat too much of it, but that seems to be a lot of things. Don’t eat too much or you get fat. But apparently, you don’t have to worry about saturated fats being bad for you.

    • Aatube
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      58 months ago

      WHO report

      someone else online summarized the genetics part as the following:

      Mandelian randomisation studies show that LDL-c is causative in atherogenic plaques 1 and metabolic ward RCTs show that SFA intakes increase LDL-c, while the decrease in SFAs lead to lower total and LDL-c 2.

      But yes, almost all nutrition science is a bit inconclusive because of genetic variation.

      • Forgive me, because I’m struggling to understand the linked information, but as someone with atherosclerosis this is an issue close to my heart (ha!).

        I just want to make sure I understand you.

        Your link to the european heart journal says that the causal link between LDL and ASCVD is “unequivocal”.

        I think the WHO study says (amongst a lot of other complicated stuff) that replacing SFAs with PUFAs and MUFAs is more favourable than replacing SFAs with complex carbohydrates? The strong implication being (although I couldn’t see this exactly) that higher SFA intake contributes to heart disease.

        • Aatube
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          38 months ago

          I don’t think it tries to compare carbohydrates to any UFAs, but the implication is indeed that SFAs significantly contribute to heart disease.

          • effects on the serum lipoprotein profile of reducing SFA intake by replacing a mixture of SFA with cis-PUFA […] or cis-MUFA […] were more favourable than replacing SFA with a mixture of carbohydrates.