Forgive me, because I’m struggling to understand the linked information, but as someone with atherosclerosis this is an issue close to my heart (ha!).
I just want to make sure I understand you.
Your link to the european heart journal says that the causal link between LDL and ASCVD is “unequivocal”.
I think the WHO study says (amongst a lot of other complicated stuff) that replacing SFAs with PUFAs and MUFAs is more favourable than replacing SFAs with complex carbohydrates? The strong implication being (although I couldn’t see this exactly) that higher SFA intake contributes to heart disease.
effects on the serum lipoprotein profile
of reducing SFA intake by replacing a mixture of SFA with cis-PUFA […] or cis-MUFA […] were more favourable than replacing SFA with a mixture of carbohydrates.
Forgive me, because I’m struggling to understand the linked information, but as someone with atherosclerosis this is an issue close to my heart (ha!).
I just want to make sure I understand you.
Your link to the european heart journal says that the causal link between LDL and ASCVD is “unequivocal”.
I think the WHO study says (amongst a lot of other complicated stuff) that replacing SFAs with PUFAs and MUFAs is more favourable than replacing SFAs with complex carbohydrates? The strong implication being (although I couldn’t see this exactly) that higher SFA intake contributes to heart disease.
I don’t think it tries to compare carbohydrates to any UFAs, but the implication is indeed that SFAs significantly contribute to heart disease.
Ah, thanks, I’ve missed that.